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Posted by randycupertino 18 hours ago

Semaglutide linked to lower predicted dementia risk(alz-journals.onlinelibrary.wiley.com)
422 points | 296 commentspage 2
overgard 14 hours ago|
Some researchers have (informally) taken to calling certain forms of dementia "type-3 diabetes", so I think this sounds at least plausible. It's far from a foregone conclusion or anything, but there's at least some evidence that dementia might be a metabolic issue, so it would make sense that something that improves metabolic health would also improve brain health.
p-o 16 hours ago||
Although it's encouraging, they do mention that the reduction in the BMI also have an effect (they take it into the consideration by lowering the β −0.092 to β −0.066 (P < 0.001)).

Just skimming through it, it's possible there are direct benefits, but it could also be other environmental factor. This study will most likely generate others that give us more insight in the future.

jimkleiber 11 hours ago||
I hope that people will study the emotional impacts of these drugs.

I wonder if that may be the root of a lot of these changes we are seeing and might provide more clues into what might be possible with them.

miki123211 15 hours ago||
I swear, Semaglutide is reverse Asbestos.

It's something we found and started using to one specific problem, but it later turned out that it has lots of other, extremely wide-ranging consequences. Except that in this case, these consequences are positive.

swalsh 4 minutes ago||
Calling it now, the downside is the effects are most apparent on higher dosages, but you cant take higher dosages forever. Eventually you run out of body fat, have go reduce the dose, and the benefits dither.
amelius 12 hours ago||
Semaglutide reduces appetite. Who could have known that eating less will have positive effects?

Maybe we should try consuming less in general and see what the effects are. Is there a semaglutide for my shopping habits? Maybe ban ads?

bitexploder 11 hours ago||
GLP-1 does a lot more than just stomach signaling. It shrinks visceral fat, it's been shown to clear liver fat, and it actually increases mitochondrial efficiency similar to if you were to do endurance work/cardio. There's a lot going on with it. That's kind of what the parent is talking about, all these second-order effects that were not obvious. There is also evidence of down-regulated mTOR signaling and epigenetic protection. And these aren't all just bullshit, big pharma funded studies either.
amelius 11 hours ago||
> It shrinks visceral fat, it's been shown to clear liver fat

The brain is 60% fat, so I'm not running to the pharmacy yet.

juiceland 10 hours ago||
Myelin and membranes aren’t triglyceride stores. There’s no lipolytic pathway that strips a neuron’s membrane the way a caloric deficit empties an adipocyte.
Alien1Being 17 hours ago||
"Semaglutide attenuates a proteomics-based dementia risk signature "

ie : clinically meaningless.

The company behind this, Novo Nordisk is increasingly desperate, as tirzepatide destroys the semaglutide revenue stream and the consequent job losses decimate the company

dghlsakjg 16 hours ago||
Their operating profit was up 11% on a margin of 44% according to their recent quarterly release, and they beat EPS by 23%. They were in a little trouble in the stock market a few years ago as competitors came online, but they are doing pretty well right now.

They have launched new semaglutide oral products which are growing faster than previous products, and they have multiple drugs in the research pipeline in late phases.

Scharkenberg 16 hours ago||
Can you elaborate on your claims? As it currently stands, I’m only reading meaningless drivel from you.
anonuser123 16 hours ago||
Read a interesting piece recently that Semaglutide reduces inflammation, and very likely the weight-loss and a lot of the other emerging benefits are likely the effects of reduced inflammation.
Aurornis 16 hours ago|
> and very likely the weight-loss and a lot of the other emerging benefits are likely the effects of reduced inflammation.

That’s not correct. GLP-1s interact with satiety (fullness) circuits and reduce appetite. They also have minor effects delaying gastric emptying, meaning the stomach stays full longer.

There is some early data suggesting that they might reduce some inflammation markers slightly more than weigh loss alone, but losing weight and controlling food intake without GLP-1s reduces the same inflammatory markers. The question now is if GLP-1s have additional anti-inflammatory influence.

I don’t know why someone would claim all of their effects are downstream of inflammation. Some people get stuck in modes where they think inflammation is the cause of everything and can’t comprehend causal effects going in the other direction.

nwienert 2 hours ago||
> The question now is if GLP-1s have additional anti-inflammatory influence.

This is well established, not at all a question.

> they might reduce some inflammation markers slightly more than weigh loss alone

There's no "slightly," It's going to be a news story for years to come. It's a broad spectrum anti-inflammatory drug of a breadth and efficacy never seen before.

fkdupman 14 hours ago||
Reading about GLP-1 medications reminds me of the movie Limitless. Being on them sounds great, getting off of them not so much.
paulpauper 14 hours ago|
not really. these drugs only merely make you not overweight or obese, which was normal until around 1980 or so.
ShinyLeftPad 13 hours ago||
The study implies it also does other stuff though. That's the thing
rimeice 16 hours ago||
in older adults *with obesity *and cardiovascular disease without diabetes
antonmks 16 hours ago||
Absolutely magical drug ! It makes you thinner, younger and now smarter !
mathgeek 14 hours ago|
Alzheimer's disease is not a reduction in intelligence, don't be crass.
enquirewithin 13 hours ago|
And a doubling in the rate of waking up blind.
juiceland 11 hours ago|
Doubling doesn’t mean much without knowing the base rate.
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